axio observer z1 inverted microscope (Carl Zeiss)
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Carl Zeiss
axio observer z1 inverted microscope
Axio Observer Z1 Inverted Microscope, supplied by Carl Zeiss, used in various techniques. Bioz Stars score: 99/100, based on 7752 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/inverted+microscope+system/Primovert+Compact+Inverted+Microscope/pm42919372-72-50-48
Average 99 stars, based on 7752 article reviews
Axio Observer Z1 Inverted Microscope, supplied by Carl Zeiss, used in various techniques. Bioz Stars score: 99/100, based on 7752 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/inverted+microscope+system/Primovert+Compact+Inverted+Microscope/pm42919372-72-50-48
Average 99 stars, based on 7752 article reviews
axio observer z1 inverted microscope - by Bioz Stars,
2026-11
99/100 stars
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Imaging:Article Title: Apoptotic dysregulation mediates stem cell competition and tissue regeneration. Article Snippet: .. Imaging of cell competition assays was performed at 5DPS, and proliferation at 3DPS using a Article Title: Apoptotic dysregulation mediates stem cell competition and tissue regeneration Article Snippet: .. Imaging of cell competition assays was performed at 5DPS, and proliferation at 3DPS using a Article Title: Luteolin Confers Cerebroprotection after Subarachnoid Hemorrhage by Suppression of NLPR3 Inflammasome Activation through Nrf2-Dependent Pathway. Article Snippet: .. Fluorescence microscopy imaging was examined under a ZEISS Inverted Microscopy:Article Title: Apoptotic dysregulation mediates stem cell competition and tissue regeneration. Article Snippet: .. Imaging of cell competition assays was performed at 5DPS, and proliferation at 3DPS using a Article Title: The Nrf2-GPX4 axis mitigates ferroptosis-driven early brain injury in experimental subarachnoid hemorrhage. Article Snippet: Subarachnoid hemorrhage (SAH) is a catastrophic cerebrovascular event often resulting from the rupture of intracranial aneurysms, leading to significant brain damage and lasting neurological deficits.. Despite advancements in treatment, the mechanisms underlying early brain injury (EBI) following SAH, particularly the role of ferroptosis, remain poorly understood.. This study aims to elucidate the impact of Nrf2glutathione peroxidase 4 (GPX4) pathway on ferroptosis in EBI after experimental SAH. Article Title: SIRT1 Promotes M2 Microglia Polarization via Reducing ROS-Mediated NLRP3 Inflammasome Signaling After Subarachnoid Hemorrhage. Article Snippet: Primary neurons were incubated with primary antibody against NeuN (1:200, cat# MAB377, EMDMillipore, USA) and caspase-1 p20 (1:50, cat# SC-398715, Santa Cruz Biotechnology) overnight at 4°C followed by incubation with proper second antibodies. .. The sections were visualized under a ZEISS Article Title: Apoptotic dysregulation mediates stem cell competition and tissue regeneration Article Snippet: .. Imaging of cell competition assays was performed at 5DPS, and proliferation at 3DPS using a Article Title: Development of an efficient capillary electrophoresis method for the analysis of salting-out assisted liquid-liquid extracted plasma samples. Article Snippet: Data acquisition and analysis were performed using 32 Karat software (Version 8.0, Beckman Coulter. .. Microscopy images and videos were captured using an Article Title: Ferroptosis in brain microvascular endothelial cells mediates blood-brain barrier disruption after traumatic brain injury. Article Snippet: Ferroptosis is a newly recognized form of regulated cell death.. Recently, growing evidence has shown that ferroptosis is involved in the pathogenesis of traumatic brain injury (TBI).. However, less attention has been paid to its role in brain microvascular endothelial cells (BMVECs) and blood-brain barrier (BBB) damage, the central pathological process in secondary brain injury of TBI. Article Title: Type H vessel/platelet-derived growth factor receptor β + perivascular cell disintegration is involved in vascular injury and bone loss in radiation-induced bone damage. Article Snippet: Funding information National Natural Science Foundation of China, Grant/Award Numbers: 81870769, 81900976, 82170960; Science and Technology Planning Project of Guangzhou, China, Grant/Award Number: 202206080009 Abstract Collapse of the microvascular system is a prerequisite for radiation-induced bone loss.. Since type H vessels, a specific bone vessel subtype surrounded by plateletderived growth factor receptor β (PDGFRβ) perivascular cells (PVCs), has been recently identified to couple angiogenesis and osteogenesis, we hypothesize that type H vessel injury initiates PDGFRβ PVC dysfunction, which contributes to the abnormal angiogenesis and osteogenesis after irradiation.. In this study, we found that radiation led to the decrease of both type H endothelial cell (EC) and PDGFRβ PVC numbers. Article Title: Luteolin Confers Cerebroprotection after Subarachnoid Hemorrhage by Suppression of NLPR3 Inflammasome Activation through Nrf2-Dependent Pathway. Article Snippet: .. Fluorescence microscopy imaging was examined under a ZEISS Fluorescence:Article Title: The Nrf2-GPX4 axis mitigates ferroptosis-driven early brain injury in experimental subarachnoid hemorrhage. Article Snippet: Subarachnoid hemorrhage (SAH) is a catastrophic cerebrovascular event often resulting from the rupture of intracranial aneurysms, leading to significant brain damage and lasting neurological deficits.. Despite advancements in treatment, the mechanisms underlying early brain injury (EBI) following SAH, particularly the role of ferroptosis, remain poorly understood.. This study aims to elucidate the impact of Nrf2glutathione peroxidase 4 (GPX4) pathway on ferroptosis in EBI after experimental SAH. Article Title: Luteolin Confers Cerebroprotection after Subarachnoid Hemorrhage by Suppression of NLPR3 Inflammasome Activation through Nrf2-Dependent Pathway. Article Snippet: .. Fluorescence microscopy imaging was examined under a ZEISS Microscopy:Article Title: The Nrf2-GPX4 axis mitigates ferroptosis-driven early brain injury in experimental subarachnoid hemorrhage. Article Snippet: Subarachnoid hemorrhage (SAH) is a catastrophic cerebrovascular event often resulting from the rupture of intracranial aneurysms, leading to significant brain damage and lasting neurological deficits.. Despite advancements in treatment, the mechanisms underlying early brain injury (EBI) following SAH, particularly the role of ferroptosis, remain poorly understood.. This study aims to elucidate the impact of Nrf2glutathione peroxidase 4 (GPX4) pathway on ferroptosis in EBI after experimental SAH. Article Title: Development of an efficient capillary electrophoresis method for the analysis of salting-out assisted liquid-liquid extracted plasma samples. Article Snippet: Data acquisition and analysis were performed using 32 Karat software (Version 8.0, Beckman Coulter. .. Microscopy images and videos were captured using an Article Title: Luteolin Confers Cerebroprotection after Subarachnoid Hemorrhage by Suppression of NLPR3 Inflammasome Activation through Nrf2-Dependent Pathway. Article Snippet: .. Fluorescence microscopy imaging was examined under a ZEISS Software:Article Title: The Nrf2-GPX4 axis mitigates ferroptosis-driven early brain injury in experimental subarachnoid hemorrhage. Article Snippet: Subarachnoid hemorrhage (SAH) is a catastrophic cerebrovascular event often resulting from the rupture of intracranial aneurysms, leading to significant brain damage and lasting neurological deficits.. Despite advancements in treatment, the mechanisms underlying early brain injury (EBI) following SAH, particularly the role of ferroptosis, remain poorly understood.. This study aims to elucidate the impact of Nrf2glutathione peroxidase 4 (GPX4) pathway on ferroptosis in EBI after experimental SAH. Staining:Article Title: Type H vessel/platelet-derived growth factor receptor β + perivascular cell disintegration is involved in vascular injury and bone loss in radiation-induced bone damage. Article Snippet: Funding information National Natural Science Foundation of China, Grant/Award Numbers: 81870769, 81900976, 82170960; Science and Technology Planning Project of Guangzhou, China, Grant/Award Number: 202206080009 Abstract Collapse of the microvascular system is a prerequisite for radiation-induced bone loss.. Since type H vessels, a specific bone vessel subtype surrounded by plateletderived growth factor receptor β (PDGFRβ) perivascular cells (PVCs), has been recently identified to couple angiogenesis and osteogenesis, we hypothesize that type H vessel injury initiates PDGFRβ PVC dysfunction, which contributes to the abnormal angiogenesis and osteogenesis after irradiation.. In this study, we found that radiation led to the decrease of both type H endothelial cell (EC) and PDGFRβ PVC numbers. |